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In the latter, cysteine originates from the reduction of cystine transported into the cell by cystine/glutamate antiporter (system Xc , xCT) [26]

In fact, transient overexpression of EAAT3 in hippocampal HT22 cells led to increased intracellular GSH levels in the presence of high glutamate concentrations, offering protection against oxidative glutamate toxicity [44, 45]

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Inhibition of DDR1-BCR signalling by nilotinib as a new therapeutic strategy for metastatic colorectal cancer